The present analysis is an extension of Spitz’s earlier (1988) study on the relationship between mental retardation (MR) lower score and subtest heritability (h2) and g-loadings. These relationships were found to be positive. But Spitz himself haven’t tested the possibility that MR (lower) score could be related with shared (c2) or nonshared (e2) environment. I use the WAIS and WISC data given in my earlier post, and have found that MR is not related with c2 and e2 values. These findings nevertheless must be interpreted very carefully because the small number of subtests (e.g., 10 or 11) is a very critical limitation.
I analyze two studies who provide the necessary data for studying the test-retest effects, namely, Watkins (2007), Schellenberg (2004, 2006). Both used the Wechsler’s subtests, and the correlations between the IQ changes among those subtests with g-loadings are negative, in line with earlier studies on this topic.
A correlation between the g factor and indices of heritability (h2) gives support for the genetic g hypothesis but, on the other hand, the interpretation may appear questionable if g correlates with shared (c2) and/or non shared (e2) environment to the same extent. The results from the present meta-analysis tend to support the hereditarian hypothesis.
Studies of the nature of the Flynn Effect are usually done in developed countries (e.g., Rushton, 1999; Wicherts, 2004; Nijenhuis, 2007; for an ‘Overview of the Flynn Effect’, see Williams, 2013). There are two recent data on two developing countries (Khaleefa, 2009; Liu, 2012). The reported numbers on subtests gains can be studied using either MCV or PC analysis. Next, we will see that shared (c²) and non-shared (e²) environments, as measured by Falconer’s formula, are unrelated to heritability (h²) of the WAIS and WISC subtests. Culture load, heritability, g-loadings, and black-white differences tend to form a common cluster (on PC1) that is different from the pattern of loadings shown by shared and non-shared environment.
The french adoption study, by Capron & Duyme (1989, Table 2; 1996, Table 3), attempted to show that IQ can be improved by adoption. Their numbers displayed an IQ gain of 15 or even 20 points (WISC-R). To recall, Jensen (1997) analyzed Capron and Duyme adoption data (1996) with N=38, a study often cited by environmentalists as evidence against the hereditarian hypothesis. In Adoption Data and Two g-Related Hypotheses, Jensen shows that IQ difference owing to the adoption of children from low-SES parents by high-SES families is not g-loaded while at the same time the IQ difference owing to low-SES versus high-SES biological families loaded in fact on the g factor or PC1. Plus, the SES-difference of adopted families correlated at only 0.099 with SES-difference of biological families.
In Bias in Mental Testing (1980, pp. 546-548), Arthur Jensen showed that a congruence coefficient test from a factor analysis of the within- (WF) and between-family (BF) correlations among blacks and whites could yield an identical g factor structure. A similarity in factorial structure for these four groups having been evidenced, he writes :
These correlations are statistically homogeneous; that is, they do not differ significantly from one another. Thus it appears that the g loadings of these seven tests show a very similar pattern regardless of whether they were extracted from the within-family correlations (which completely exclude cultural and socioeconomic effects in the factor analyzed variance) or from the between-families correlations, for either whites or blacks. … This outcome would seem unlikely if the largest source of variance in these tests, reflected by their g loadings, were strongly influenced by whatever cultural differences that might exist between families and between whites and blacks.
Jensen (1980, Table 4) has been replicated by Nagoshi and Johnson (1987, pp. 310-314). I will replicate those earlier tests using NLSY97 and NLSY79. As Jensen (1998, pp. 99-100) noted, the congruence coefficient (CC) can be interpreted as being an index of factor similarity.
At what age does the cognitive ability gap between blacks and whites first appear? At what age does the black-white ability gap stop growing?
Knowing the answers to these questions is vital to understanding the etiology of the black-white ability gap, especially if this gap has an environmental cause. However, the only scholarly work that attempts to investigate these issues is John Loehlin’s Race Differences in Intelligence (1975), which is nearly 40 years old. So I will update and expand upon that review here on Human Varieties by summarizing all available measurements of African American cognitive ability from early infancy to age 3; I will also discuss the relevance of this data to current debates in the social sciences.